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We characterized cerebral infarction in children with tuberculous meningitis and explored its relationship with systemic inflammatory mediators using targeted transcriptomic analysis. Children with tuberculous meningitis had baseline magnetic resonance imaging scans and whole blood RNA sequencing for matrix metalloproteinases (MMP-8, MMP-9, TIMP-1), cytokines (IL-10, IL-1β, TNF-α, IFN-γ), and growth factors (VEGF). Overall 22 (73%) children had mild disease and 19 (63%) had cerebral infarctions, which were commonly acute (n = 9, 47%), multiple (n = 14, 74%), and bilateral (n = 12, 63%), occurring in cerebral hemispheres (n = 12, 59%), basal ganglia (n = 10, 53%), and thalamus (n = 5, 26%). Children with infarctions had significantly higher cerebrospinal fluid protein, lower cerebrospinal fluid glucose, and higher systemic MMP-8 expression.

Original publication

DOI

10.1093/infdis/jiaf399

Type

Journal

The Journal of infectious diseases

Publication Date

10/2025

Volume

232

Pages

e676 - e681

Addresses

Centre for Tropical Medicine and Global Health, Nuffield Department of Medicine, Oxford University, Oxford, United Kingdom.

Keywords

Brain, Humans, Tuberculosis, Meningeal, Cerebral Infarction, Cytokines, Magnetic Resonance Imaging, Gene Expression Profiling, Adolescent, Child, Child, Preschool, Infant, Female, Male, Matrix Metalloproteinase 9, Transcriptome